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urine albumin elisa kit  (Bethyl)


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    Bethyl urine albumin elisa kit
    Urine Albumin Elisa Kit, supplied by Bethyl, used in various techniques. Bioz Stars score: 93/100, based on 102 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/urine+albumin/Bovine+Albumin+ELISA+Kit/pm41056767-80-8-13
    Average 93 stars, based on 102 article reviews
    urine albumin elisa kit - by Bioz Stars, 2026-10
    93/100 stars

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    other:

    Article Title: LINC00355 Mediates CTNNBIP1 Promoter Methylation and Promotes Endoplasmic Reticulum Stress-Induced Podocyte Injury in Diabetic Nephropathy.
    Article Snippet: Aims: Endoplasmic reticulum stress (ER stress) plays an important role in podocyte injury in diabetic nephropathy.. Wnt/b-catenin signaling modulates ER stress, yet the epigenetic regulation of b-catenin in ER stress and podocyte injury remains largely unknown.. Herein, we tested the hypothesis that LINC00355 recruits EZH1 to the promoter region of CTNNBIP1 and trimethylates H3K4 to regulate ER-stress induced podocyte injury in DN.

    Enzyme-linked Immunosorbent Assay:

    Article Title: Exacerbation of diabetes due to F. Nucleatum LPS-induced SGLT2 overexpression in the renal proximal tubular epithelial cells.
    Article Snippet: .. The samples were analyzed for urine albumin by albumin ELISA kit (Bethyl Laboratories, Inc. Montgomery, Texas, USA), for blood urea nitrogen (BUN) by DetectX (BUN detection kit, Arbor Assays LLC, Ann Arbor, MI, USA), and for blood creatinine (CRE) by LabAssay (Fujifilm Wako Pure Chemical Corporation, Osaka, Japan). ..

    Article Title: Quinolinic acid potentially links kidney injury to brain toxicity
    Article Snippet: Mouse CCL2/JE/MCP-1 Quantikine ELISA Kit (R&D, #MJE00B), Human/Mouse Total HIF-1 alpha/HIF1A DuoSet IC ELISA (R&D, #DYC1935-2), Mouse IL-6 Quantikine ELISA Kit (R&D, #M6000B-1). .. For urine Albumin-to-Creatinine-Ratio (ACR): Mouse Albumin ELISA Kit (Bethyl Laboratories, #E99-134), and Creatinine Colorimetric Detection Kit (ENZO; #ADI-907-030A). ..

    Article Title: The Abnormal Expression of Tubular SGLT2 and GULT2 in Diabetes Model Mice with Malocclusion-Induced Hyperglycemia
    Article Snippet: .. The samples were analyzed for urine albumin by albumin ELISA kit (Bethyl Laboratories, Inc., Montgomery, TX, USA), for blood urea nitrogen (BUN) by DetectX (BUN detection kit, Arbor Assays LLC, Ann Arbor, MI, USA), and for blood creatinine (CRE) by LabAssay (Fujifilm Wako Pure Chemical Corporation, Osaka, Japan). .. For the mRNA expression amounts of SGLT2 and GLUT2 in the mouse kidney tissue and of TNF-α and interleukin (IL)-6 in the mouse gingival tissue around the teeth with mal-occlusion and large intestine, the real-time PCR was conducted by primer sets where the specificities had been confirmed by the manufacturer (Sigma-Aldrich Japan) described elsewhere ( ) [ ].

    Article Title: RTN1A mediates diabetes-induced AKI-to-CKD transition
    Article Snippet: .. Urine albumin was quantified by a commercially available ELISA kit (Bethyl Laboratories, E99-134), and creatinine levels were measured in the same samples using a creatinine assay kit (BioAssay Systems, DICT-500). ..

    Article Title: Exacerbation of diabetes due to F. Nucleatum LPS-induced SGLT2 overexpression in the renal proximal tubular epithelial cells
    Article Snippet: .. The samples were analyzed for urine albumin by albumin ELISA kit (Bethyl Laboratories, Inc. Montgomery, Texas, USA), for blood urea nitrogen (BUN) by DetectX (BUN detection kit, Arbor Assays LLC, Ann Arbor, MI, USA), and for blood creatinine (CRE) by LabAssay (Fujifilm Wako Pure Chemical Corporation, Osaka, Japan). ..

    Article Title: RTN1A mediates diabetes-induced AKI-to-CKD transition.
    Article Snippet: .. Urine albumin was quantified by a commercially available ELISA kit (Bethyl Laboratories, E99-134), and creatinine levels were measured in the same samples using a creatinine assay kit (BioAssay Systems, DICT-500). ..

    Article Title: The Abnormal Expression of Tubular SGLT2 and GULT2 in Diabetes Model Mice with Malocclusion-Induced Hyperglycemia.
    Article Snippet: .. The samples were analyzed for urine albumin by albumin ELISA kit (Bethyl Laboratories, Inc., Montgomery, TX, USA), for blood urea nitrogen (BUN) by DetectX (BUN detection kit, Arbor Assays LLC, Ann Arbor, MI, USA), and for blood creatinine (CRE) by LabAssay (Fujifilm Wako Pure Chemical Corporation, Osaka, Japan). .. For the mRNA expression amounts of SGLT2 and GLUT2 in the mouse kidney tissue and of TNF-α and interleukin (IL)-6 in the mouse gingival tissue around the teeth with mal-occlusion and large intestine, the real-time PCR was conducted by primer sets where the specificities had been confirmed by the manufacturer (Sigma-Aldrich Japan) described elsewhere (Table 1) [13].



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    Fig. 5 In vivo therapeutic effects of Dex@NBs-TRPC6. (A) Schematic diagram of in vivo treatment and therapy protocol. Normal <t>mouse</t> without any treat ment was dedicated as control group. (B) Albuminuria <t>(urine</t> <t>albumin-to-creatinine</t> ratio) was determined in control group, ADR + NS group and three Dex-treated ADR groups. n = 9–10 mice per group. (C) Representative kidney images of PAS staining. Upper: glomerular morphology, scale bar = 20 μm; Lower: tubule-interstitial morphology, scale bar = 50 μm. (D) Quantitative analysis of glomerulosclerosis severity. (E) Quantitative analysis of tubular injury. (F) RT-PCR analysis of mRNA expression of Kim-1. (G) RT-PCR analysis of mRNA expression of NGAL. n = 9. (H) Representative images of TEM of glomerular basement membrane. Top: scale bar = 2 μm, Bottom: scale bar = 500 nm. (I) Quantitative analysis of foot process width. n = 6 mice per group in all histo logical analysis. (J) Upper: immunofluorescence staining of WT1 expressed in nuclei of mouse podocytes, scale bar = 20 μm. Lower: immunofluorescence staining of podocin expressed along the glomerular basement membrane, scale bar = 20 μm. (K) Quantitative analysis of WT1 positive cell numbers per glomerulus, n = 6. (L) Quantitative analysis of fluorescence intensity of podocin, n = 6. Data are presented as mean ± SEM, one-way ANOVA. *p < 0.05, **p < 0.01, vs. Control group, #p < 0.05, ##p < 0.01, vs. ADR + NS group, $p < 0.05, $$p < 0.01 as indicated
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    Fig. 5 In vivo therapeutic effects of Dex@NBs-TRPC6. (A) Schematic diagram of in vivo treatment and therapy protocol. Normal <t>mouse</t> without any treat ment was dedicated as control group. (B) Albuminuria <t>(urine</t> <t>albumin-to-creatinine</t> ratio) was determined in control group, ADR + NS group and three Dex-treated ADR groups. n = 9–10 mice per group. (C) Representative kidney images of PAS staining. Upper: glomerular morphology, scale bar = 20 μm; Lower: tubule-interstitial morphology, scale bar = 50 μm. (D) Quantitative analysis of glomerulosclerosis severity. (E) Quantitative analysis of tubular injury. (F) RT-PCR analysis of mRNA expression of Kim-1. (G) RT-PCR analysis of mRNA expression of NGAL. n = 9. (H) Representative images of TEM of glomerular basement membrane. Top: scale bar = 2 μm, Bottom: scale bar = 500 nm. (I) Quantitative analysis of foot process width. n = 6 mice per group in all histo logical analysis. (J) Upper: immunofluorescence staining of WT1 expressed in nuclei of mouse podocytes, scale bar = 20 μm. Lower: immunofluorescence staining of podocin expressed along the glomerular basement membrane, scale bar = 20 μm. (K) Quantitative analysis of WT1 positive cell numbers per glomerulus, n = 6. (L) Quantitative analysis of fluorescence intensity of podocin, n = 6. Data are presented as mean ± SEM, one-way ANOVA. *p < 0.05, **p < 0.01, vs. Control group, #p < 0.05, ##p < 0.01, vs. ADR + NS group, $p < 0.05, $$p < 0.01 as indicated
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    Fig. 5 In vivo therapeutic effects of Dex@NBs-TRPC6. (A) Schematic diagram of in vivo treatment and therapy protocol. Normal mouse without any treat ment was dedicated as control group. (B) Albuminuria (urine albumin-to-creatinine ratio) was determined in control group, ADR + NS group and three Dex-treated ADR groups. n = 9–10 mice per group. (C) Representative kidney images of PAS staining. Upper: glomerular morphology, scale bar = 20 μm; Lower: tubule-interstitial morphology, scale bar = 50 μm. (D) Quantitative analysis of glomerulosclerosis severity. (E) Quantitative analysis of tubular injury. (F) RT-PCR analysis of mRNA expression of Kim-1. (G) RT-PCR analysis of mRNA expression of NGAL. n = 9. (H) Representative images of TEM of glomerular basement membrane. Top: scale bar = 2 μm, Bottom: scale bar = 500 nm. (I) Quantitative analysis of foot process width. n = 6 mice per group in all histo logical analysis. (J) Upper: immunofluorescence staining of WT1 expressed in nuclei of mouse podocytes, scale bar = 20 μm. Lower: immunofluorescence staining of podocin expressed along the glomerular basement membrane, scale bar = 20 μm. (K) Quantitative analysis of WT1 positive cell numbers per glomerulus, n = 6. (L) Quantitative analysis of fluorescence intensity of podocin, n = 6. Data are presented as mean ± SEM, one-way ANOVA. *p < 0.05, **p < 0.01, vs. Control group, #p < 0.05, ##p < 0.01, vs. ADR + NS group, $p < 0.05, $$p < 0.01 as indicated

    Journal: Journal of nanobiotechnology

    Article Title: TRPC6-targeted dexamethasone nanobubbles with ultrasound-guided theranostics for adriamycin-induced nephropathy.

    doi: 10.1186/s12951-025-03487-8

    Figure Lengend Snippet: Fig. 5 In vivo therapeutic effects of Dex@NBs-TRPC6. (A) Schematic diagram of in vivo treatment and therapy protocol. Normal mouse without any treat ment was dedicated as control group. (B) Albuminuria (urine albumin-to-creatinine ratio) was determined in control group, ADR + NS group and three Dex-treated ADR groups. n = 9–10 mice per group. (C) Representative kidney images of PAS staining. Upper: glomerular morphology, scale bar = 20 μm; Lower: tubule-interstitial morphology, scale bar = 50 μm. (D) Quantitative analysis of glomerulosclerosis severity. (E) Quantitative analysis of tubular injury. (F) RT-PCR analysis of mRNA expression of Kim-1. (G) RT-PCR analysis of mRNA expression of NGAL. n = 9. (H) Representative images of TEM of glomerular basement membrane. Top: scale bar = 2 μm, Bottom: scale bar = 500 nm. (I) Quantitative analysis of foot process width. n = 6 mice per group in all histo logical analysis. (J) Upper: immunofluorescence staining of WT1 expressed in nuclei of mouse podocytes, scale bar = 20 μm. Lower: immunofluorescence staining of podocin expressed along the glomerular basement membrane, scale bar = 20 μm. (K) Quantitative analysis of WT1 positive cell numbers per glomerulus, n = 6. (L) Quantitative analysis of fluorescence intensity of podocin, n = 6. Data are presented as mean ± SEM, one-way ANOVA. *p < 0.05, **p < 0.01, vs. Control group, #p < 0.05, ##p < 0.01, vs. ADR + NS group, $p < 0.05, $$p < 0.01 as indicated

    Article Snippet: Mouse urine albumin concentration was measured using an ELISA kit (Bethyl Laboratories.

    Techniques: In Vivo, Control, Staining, Reverse Transcription Polymerase Chain Reaction, Expressing, Membrane, Immunofluorescence, Fluorescence